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The underlying protective mechanisms of ALA in renal ischemia reperfusion injury may be multifactorial, including counteracting oxidative damage [32,75] and downregulation of channels, enzymes, and transporters, such as aquaporins and sodium transporters, as well as sodium-potassium ATPase and nitric oxide synthase isoforms [78]

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Reactive oxygen species (ROS)-responsive size-reducible nanoassemblies for deeper atherosclerotic plaque penetration and enhanced macrophage-targeted drug delivery